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Among positive regulators, TGFβ, insulin-like growth factor I, estrogens and 1.25-OH 2 D 3 have been reported to increase the transcription and/or the activity of LOX.6,7,8 In contrast, homocysteine is believed to negatively affect LOX expression and activity.9 It was also demonstrated that homocysteine binds to the aldehyde groups of allysine and hydroxylysine, which are the precursors of enzymatic crosslinks and leads to a marked decrease in their levels.10 Other factors such as fibroblast growth factor and high dose of PGE2 and TNFα also inhibit LOX expression.11,12 Recently it has been demonstrated that the gamma-carboxyglutamic acid containing non-collagenous protein, periostin, has a major role in modulating the activity of LOX.13 Indeed LOX is synthesized as a non-active precursor, proLOX, which is activated by the cleavage of the propeptide mediated by BMP1, which is a procollagen peptidase
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Metagenomic characterization of gut microbiota of carriers of extended-spectrum beta-lactamase or carbapenemase-producing Enterobacteriaceae following treatment with oral antibiotics and fecal microbiota transplantation: results from a multicenter randomized trial
